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Wild Animals Don’t Count Calories or Sign Up for Step Aerobics

Have you ever seen an obese wild ani­mal? Look at these wilde­beests in Krüger Nation­al Park in South Africa. There’s no cel­lulite on their thighs! Wilde­beest weigh only 40 pounds at birth, but then they gain weight rapid­ly. By the time they’re a year old, they weigh about 200 pounds. The females reach a peak weight of about 350 pounds at 4 years of age. The males peak at 500 pounds at 5 years of age. Yet after that, their weight stays remark­ably sta­ble. Why do they stop gain­ing weight? Since they don’t start count­ing calo­ries or tak­ing step aer­o­bics class­es in adult­hood, they must have some built-in mech­a­nisms that reg­u­late their weight nat­u­ral­ly. Do humans also have in-born weight-con­trol mech­a­nisms? If so, why have so many peo­ple been get­ting so fat late­ly?

To keep our body weight at a nor­mal lev­el, we are told to engage in unnat­ur­al behav­iors. We’re told to eat less and move more. Yet wild ani­mals nev­er lim­it their food por­tions, and they do only the amount of activ­i­ty they feel like doing. I think that their secret for stay­ing slim is that they eat the kind of food that is appro­pri­ate for their species. If you trapped some wilde­beest in a pen and fed them a diet that was much rich­er in calo­ries than what they ate in the wild, they’d prob­a­bly get fat. That’s what has hap­pened to human beings in indus­tri­al­ized soci­eties. To cure our weight prob­lems, we need to escape from our cubi­cles and start eat­ing a more nat­ur­al diet. Go play out­side, and eat low-fat unre­fined plant foods instead of eat­ing ani­mals and processed foods.

wild-animals

When you look at pop­u­la­tions all over the world, you’ll notice that the peo­ple who eat a diet based on unre­fined plant foods stay nat­u­ral­ly slim and remark­ably free of heart dis­ease and dia­betes and oth­er chron­ic dis­eases. For many gen­er­a­tions, most of the world’s pop­u­la­tion ate like that. Only the rich could afford to eat large serv­ings of rich foods, such as meats and but­ter and hon­ey, on a reg­u­lar basis. As a result, only rich peo­ple suf­fered from obe­si­ty, gout, and ath­er­o­scle­ro­sis. Because of agri­cul­tur­al poli­cies, those foods have now become cheap while fresh fruit and veg­eta­bles are still rel­a­tive­ly expen­sive. As a result, the “dis­eases of afflu­ence” are now a par­tic­u­lar prob­lem for poor peo­ple in the Unit­ed States.

Pho­to by h.koppdelaney


Behind Barbed Wire_PrintNote: In my book Thin Dia­betes, Fat Dia­betes: Pre­vent Type 1, Cure Type 2, you can learn more about how a low-fat, high-fiber, high-car­bo­hy­drate diet helps peo­ple lose weight and revers­es their type 2 dia­betes.

Can Hot Chili Peppers Help Prevent Cancer?

Cap­saicin, which is the chem­i­cal that puts the heat in hot chili pep­pers, may encour­age some kinds of can­cer cells to com­mit sui­cide; but cap­saicin doesn’t seem to have the same effect on healthy cells. This is just one of many ways in which chem­i­cals that are found nat­u­ral­ly in plants (phy­to­chem­i­cals) could have an anti­cancer effect.

Can­cer isn’t just one dis­ease. It’s a group of unre­lat­ed dis­eases that all result from the same sort of prob­lem: cells behav­ing bad­ly. Can­cer cells don’t become the kind of cell that they’re sup­posed to become, and they keep divid­ing to make new cells long after they were sup­posed to stop. Some­times, they trav­el through the body and set­tle down in places where they’re not sup­posed to be. All of these prob­lems result from some­thing going wrong in the cell’s genet­ic mate­r­i­al. Either some genes have been dam­aged or the switch­es that are sup­posed to turn the genes on and off have been stuck in the wrong posi­tion. This prob­lem can get start­ed if a cell’s genes are dam­aged by expo­sure to radi­a­tion or to can­cer-caus­ing (car­cino­genic) chem­i­cals, such as those in tobac­co smoke. The first line of defense against can­cer is to reduce the body’s expo­sure to radi­a­tion and oth­er car­cino­gens.

Even after a cell has gone rogue, the body has sev­er­al lev­els of defens­es that could stamp out the can­cer before it is ever noticed. The first is a self-destruct mech­a­nism that is built into the cell’s genet­ic instruc­tions. This self-destruc­tion, which is called apop­to­sis or pro­grammed cell death, caus­es the cell to break apart into tidy frag­ments that are quick­ly and eas­i­ly devoured by white blood cells. In con­trast, when cells die as a result of trau­ma, they make a mess by spilling their con­tents into the sur­round­ing flu­id.

Pro­grammed cell death plays an impor­tant role in sculpt­ing the embryo dur­ing ear­ly devel­op­ment. If cells are in the wrong place at the wrong time, they get a sig­nal to com­mit sui­cide. That’s why most peo­ple aren’t born with webbed fin­gers and toes. Even in a healthy adult, tens of bil­lions of cells under­go pro­grammed cell death every day. Pro­grammed cell death is a nat­ur­al body process that is sup­posed to stay in a healthy bal­ance. If too many cells die, the result is tis­sue shrink­age (atro­phy). If too many cells fail to com­mit sui­cide, then abnor­mal cells such as can­cer cells can get out of con­trol.

Pro­grammed cell death is a com­pli­cat­ed process that can involve sev­er­al dif­fer­ent path­ways and that can be stim­u­lat­ed or sup­pressed by many dif­fer­ent sig­nals. How­ev­er, the end result is always the same: enzymes called cas­pas­es are acti­vat­ed, and they break down the pro­tein struc­tures inside the cell. Some kinds of can­cer cells fail to under­go pro­grammed cell death because they have a defi­cien­cy of or defect in one of their cas­pas­es. These cells may need a lit­tle extra encour­age­ment to under­go pro­grammed cell death.

Researchers have report­ed that cap­saicin inhibits the growth of colon tumors. The­o­ret­i­cal­ly, cap­saicin could have anti­tu­mor effects in oth­er parts of the body because it is eas­i­ly absorbed from the intes­tine and car­ried through­out the body by the blood­stream. One study showed that cap­saicin pro­motes pro­grammed cell death in a par­tic­u­lar type of liv­er can­cer cells. Anoth­er study showed a sim­i­lar effect in breast can­cer cells.

The first line of defense against can­cer is to avoid radi­a­tion, car­cino­genic chem­i­cals, and the viral infec­tions that are known to cause cells to become malig­nant. The sec­ond line of defense is to eat a low-fat, high-fiber, plant-based diet, which acts in sev­er­al dif­fer­ent ways to pre­vent cells from becom­ing malig­nant and to sup­press the growth of tumors. The pro­mo­tion of pro­grammed cell death by hot pep­pers is just one of the ways in which a plant-based diet could help to sup­press can­cer.

Institute of Medicine Questions Scientific Need for Chimpanzee Research

Pho­to: Chim­panzee being used for space research by the U.S. Air Force in the 1960s.

The Insti­tute of Med­i­cine con­vened an ad hoc com­mit­tee to answer two impor­tant ques­tions:

  • Is bio­med­ical research with chim­panzees “nec­es­sary for research dis­cov­er­ies and to deter­mine the safe­ty and effi­ca­cy of new pre­ven­tion or treat­ment strate­gies?”
  • Is behav­ioral research using chim­panzees “nec­es­sary for progress in under­stand­ing social, neu­ro­log­i­cal and behav­ioral fac­tors that influ­ence the devel­op­ment, pre­ven­tion, or treat­ment of dis­ease?”

The com­mit­tee was asked to con­sid­er only sci­en­tif­ic ques­tions, not ques­tions relat­ed to ethics or costs. The committee’s report con­clud­ed that most cur­rent use of chim­panzees for bio­med­ical research is unnec­es­sary and that the Nation­al Insti­tutes of Health should put strict lim­its on the use of chim­panzees as research sub­jects. The NIH has already announced a freeze on new grants for chim­panzee exper­i­men­ta­tion.

Some mem­bers of Con­gress want to out­law all exper­i­men­ta­tion on great apes, includ­ing chim­panzees (H.R. 1513: The Great Ape Pro­tec­tion and Cost Sav­ings Act).


Update: H.R. 1513 was not enact­ed.

Does Tofurkey Subliminally “Glorify” Meat-Eating?

David Siro­ta wrote an arti­cle that sug­gests that the veg­e­tar­i­an prod­ucts that mim­ic meat prod­ucts under­mine veg­e­tar­i­an­ism by glo­ri­fy­ing the con­sump­tion of meat. I had to laugh because I hon­est­ly couldn’t imag­ine Tofurkey glo­ri­fy­ing any­thing. Nor do I think that rice milk glo­ri­fies cow’s milk or that a tofu scram­ble glo­ri­fies eggs. Yet the use of these foods does raise two impor­tant nutri­tion-relat­ed ques­tions: What kind of diet is tru­ly healthy for a human being, and how can we help peo­ple find sat­is­fac­tion and delight from a tru­ly healthy diet?

Many veg­e­tar­i­ans depend heav­i­ly on the soy fake meats and “cheezes” because they are wor­ried about get­ting enough pro­tein in their diet. In real­i­ty, you don’t need to eat fake meat or cheeze to get enough pro­tein. It’s prac­ti­cal­ly impos­si­ble to find real cas­es of pro­tein defi­cien­cy in peo­ple who were get­ting enough calo­ries from any rea­son­able plant-based diet. To find cas­es of pure pro­tein defi­cien­cy, you have to look at peo­ple who have been fed noth­ing but glu­cose intra­venous­ly, or peo­ple who have a diges­tive or meta­bol­ic dis­ease, or babies who were fed some bizarre sub­sti­tute for breast milk.

Plants pro­vide all of the nutri­ents that are essen­tial for human nutri­tion, except for vit­a­min D and vit­a­min B12. Your body makes its own sup­ply of vit­a­min D if you go out in the sun­shine, and vit­a­min B12 comes from bac­te­ria. So there’s no nutri­tion­al need to include ani­mal-based food in the diet. On the con­trary, the less ani­mal-based food a pop­u­la­tion eats, the low­er its rates of death from heart dis­ease, can­cer, dia­betes, and oth­er chron­ic dis­eases tend to be.

So what about the refined plant-based foods that resem­ble ani­mal foods? Do they pose the same health threats as real ani­mal-based foods? The answer is a bit com­pli­cat­ed. The health threats that they could pose depend on how close­ly they resem­ble the ani­mal-based foods they replace.

Ani­mal-based foods con­tain fat and cho­les­terol but no fiber. No veg­an prod­ucts con­tain any cho­les­terol, but some of them do con­tain a lot of fat and lit­tle or no fiber. Thus, they could pro­mote weight gain and high cho­les­terol lev­els. Pota­to chips are veg­an; but because of all that fat and salt, they’re almost as bad for you as pork rinds.

Ani­mal-based foods con­tain far more pro­tein than you need. This excess pro­tein puts a strain on the liv­er and kid­neys. The “high-qual­i­ty” pro­tein in dairy prod­ucts, in par­tic­u­lar, also caus­es the liv­er to release a pow­er­ful growth hor­mone (IGF-1) that pro­motes the growth of can­cers. Huge serv­ings of soy pro­tein also pro­mote the secre­tion of IGF-1, but to a less­er extent than dairy prod­ucts do.

The pro­teins in ani­mal-based foods are sim­i­lar to but not exact­ly like the pro­teins in the human body. If they find their way into the blood­stream before they are com­plete­ly bro­ken down, they may cause the immune sys­tem to pro­duce anti­bod­ies that go on to attack the body’s own tis­sues. A switch to a plant-based diet can dra­mat­i­cal­ly reduce this risk. How­ev­er, some of the fake ani­mal prod­ucts are based on wheat gluten, which can cause autoim­mune prob­lems in a small per­cent­age of the pop­u­la­tion. For this rea­son, peo­ple with celi­ac dis­ease or oth­er wheat sen­si­tiv­i­ty can­not eat sei­tan.

Real meats and cheeses are high in fat but devoid of starch. The fake stuff also tends to be high in fat and low in starch. All fats are fat­ten­ing, and some of the fats from plant sources are par­tic­u­lar­ly pow­er­ful pro­mot­ers of can­cer. The plant-based diets that are tru­ly good for human health are high in fiber and starch and low in fat.

Ani­mals have hor­mones that are very much like our own. When peo­ple eat ani­mal foods, they get a dose of these hor­mones, even if the ani­mals were raised organ­i­cal­ly. Plants have dif­fer­ent hor­mones. Some plants con­tain phy­toe­stro­gens, which are sub­stances that have some sort of effect on estro­gen recep­tors. How­ev­er, some of the phy­toe­stro­gens are estro­gen block­ers or weak estro­gens that com­pete with the body’s nat­ur­al estro­gens, thus decreas­ing the effects that our native estro­gen has on our tis­sues.

Ani­mals absorb tox­ins from their envi­ron­ment and store them in their fat­ty tis­sue. That’s why it’s good to eat “low on the food chain.” The processed fake meats and cheezes are low on the food chain, but you may have to con­sid­er what kinds of addi­tives are in them.

Many peo­ple advo­cate the use of the fake meats and cheezes sort of as train­ing wheels to help peo­ple adjust to a plant-based diet. My con­cern with that approach is that these foods can be unsat­is­fy­ing because they don’t nec­es­sar­i­ly taste like the real thing. Rather than serv­ing a food that is a dim echo of some­thing else, why not serve some­thing that can stand on its own? Why eat an unsat­is­fy­ing soy pat­ty when you could eat a gen­uine bean bur­ri­to?

I use a lit­tle bit of tofu or soy milk now and then. The “fake meat” that I use exten­sive­ly is mush­rooms and nutri­tion­al yeast. I make a gar­licky low-fat mush­room gravy and serve it over huge mounds of mashed pota­toes. I add either mush­rooms or nutri­tion­al yeast to hearty stews, and nobody cares that I didn’t use a ham­bone.Pho­to by Andrea_Nguyen

How LDL Cholesterol Becomes Atherosclerotic Plaque

Here’s an inter­est­ing arti­cle about how high lev­els of LDL cho­les­terol end up caus­ing ath­er­o­scle­ro­sis.

The body uses cho­les­terol for var­i­ous pur­pos­es, and it has a sys­tem for trans­port­ing cho­les­terol in the blood­stream. Like fat, cho­les­terol doesn’t dis­solve in water. To trans­port fat and cho­les­terol via the blood­stream, the body puts them in pack­ages called lipopro­teins. As the name sug­gests, a lipopro­tein con­tains fat­ty sub­stances (lipids) as well as some pro­tein.

Not only do fat­ty sub­stances like cho­les­terol fail to dis­solve in water, they float on top of it. That’s because they are less dense than water. They are also less dense than pro­tein. The lipopro­tein par­ti­cles that are largest and con­tain the most fat also have the low­est den­si­ty. The “bad” cho­les­terol that peo­ple talk about is low-den­si­ty lipopro­tein (LDL). These are lipopro­tein par­ti­cles that car­ry fat and cho­les­terol from the liv­er to the rest of the body. LDL is like a wheel­bar­row full of fat and cho­les­terol trav­el­ing from the liv­er to the rest of the body. In con­trast, high-den­si­ty lipopro­tein (HDL) picks up the cho­les­terol from the tis­sues and car­ries it back to the liv­er. HDL is like a most­ly emp­ty wheel­bar­row pick­ing up fat and cho­les­terol and tak­ing it back to the liv­er.

The arti­cle explains that cho­les­terol is always enter­ing and leav­ing the inti­mal lay­er of the arte­r­i­al wall. The cho­les­terol is brought in by LDL and is tak­en away by HDL. If the cho­les­terol is brought in faster than it leaves, it builds up to form a deposit called an athero­ma. The more LDL there is in the blood­stream, the faster the LDL par­ti­cles enter the wall of the arter­ies. The cho­les­terol is like­ly to build up into an athero­ma if there isn’t enough HDL to car­ry the cho­les­terol back out fast enough or if the LDL under­goes some chem­i­cal change with­in the wall of the artery before it can be removed. Here’s an arti­cle that explains the kinds of chem­i­cal changes that can occur to the LDL while its inside the arte­r­i­al wall.

Why does cho­les­terol build up in the inti­ma of the arte­r­i­al wall but not in oth­er kinds of tis­sue? It’s because the con­cen­tra­tion of LDL is far high­er in the arte­r­i­al inti­ma than in any oth­er tis­sue. The prob­a­ble rea­son for this high LDL con­cen­tra­tion is the fact that the arte­r­i­al inti­ma is not drained by lymph ves­sels. LDL par­ti­cles are small enough to leak through the gaps between the endothe­lial cells that pave the inner sur­face of the artery. Then, they can dif­fuse through­out the loose struc­ture of the arte­r­i­al inti­ma. How­ev­er, they are too big to leak through the pores in the car­bo­hy­drate-and-pro­tein mesh­work of the medi­al lay­er. Thus, they can­not make their way through to the lym­phat­ic sys­tem, which is high­ly effi­cient at car­ry­ing lipopro­teins back to the blood­stream.
Pho­to by Ore­gon State Uni­ver­si­ty

To Protect Your Feet, Cure Your Type 2 Diabetes

Dia­betes is the num­ber 1 cause of non­trau­mat­ic ampu­ta­tions in the Unit­ed States. What’s tru­ly out­ra­geous is that most of these ampu­ta­tions are hap­pen­ing to peo­ple with the form of dia­betes that can eas­i­ly be cured, some­times with­in as lit­tle as a week, by a sim­ple change in diet. Just eat unre­fined plant foods instead of ani­mal-based foods and processed foods and cut your fat intake to 10% or less of calo­ries.

Type 2 dia­betes mel­li­tus is cured by remov­ing the cause, which is the fat­ty, low-fiber, stan­dard Amer­i­can diet. Peo­ple who switch to a high-fiber, low-fat (~10% of calo­ries), high-car­bo­hy­drate (75% of calo­ries), pure­ly plant-based (veg­an) diet become undi­a­bet­ic with­in a sur­pris­ing­ly short time. (They can get even quick­er results if they also start exer­cis­ing.) A prop­er diet can even relieve the ago­niz­ing pain and dan­ger­ous numb­ness from dia­bet­ic neu­ropa­thy in the feet with­in a mat­ter of days to weeks.

Only a Small Percentage of Diabetes Cases Are Truly Genetic

Late­ly, genes have been all the rage in med­ical research. Many peo­ple hope that genet­ic stud­ies will unrav­el the secrets of many dis­eases that, strict­ly speak­ing, aren’t real­ly genet­ic. Some­times, peo­ple get so caught up in think­ing about the genes that could be involved in a dis­ease that they ignore the impor­tance of diet to pre­vent and man­age the dis­ease. Yet even for a dis­ease that is tru­ly 100% genet­ic, dietary man­age­ment can be impor­tant. The clas­sic exam­ples are the rare forms of dia­betes mel­li­tus that real­ly are genet­ic. These forms of dia­betes are called mono­genic dia­betes mel­li­tus. Some­times they’re called MODY, which stands for matu­ri­ty-onset dia­betes of the young. It is a form of mono­genic dia­betes, which means that it is due to a muta­tion in a sin­gle gene.

There are sev­er­al dif­fer­ent forms of MODY, each result­ing from the muta­tion of a dif­fer­ent gene. These dis­or­ders tend to be auto­so­mal dom­i­nant, which means that if you inher­it the gene from one of your par­ents, you have the dis­ease. Clin­i­cal­ly, cas­es of MODY tend to look like a hybrid between type 1 and type 2 dia­betes. Togeth­er they account for some­where between 1% and 5% of cas­es of dia­betes mel­li­tus.

Dia­betes mel­li­tus refers to sev­er­al unre­lat­ed con­di­tions that all result in high lev­els of sug­ar in the blood­stream. In the 1870s, French researchers noticed that there are two main types of dia­betes mel­li­tus. One is a cat­a­stroph­ic dis­ease that tends to strike chil­dren and young adults. Before the dis­cov­ery of insulin, these patients would rapid­ly lose weight, go into a coma, and die with­in a mat­ter of days or weeks, regard­less of treat­ment. The French researchers called it dia­bète mai­gre, or thin dia­betes. Eng­lish-speak­ers called it juve­nile dia­betes, then insulin-depen­dent dia­betes, and now type 1 dia­betes. In con­trast, the most com­mon form of dia­betes is a much milder, reversible con­di­tion that occurs in mid­dle-aged to elder­ly adults who are usu­al­ly at least a bit over­weight. The French called this con­di­tion dia­bète gras, or fat dia­betes. Eng­lish-speak­ers tact­ful­ly referred to this kind of dia­betes as adult-onset, then non–insulin-dependent, and now type 2 dia­betes.

bothproblems

If left untreat­ed, all forms of dia­betes mel­li­tus result in abnor­mal­ly high lev­els of a sug­ar called glu­cose in the blood­stream. In cas­es of type 1 dia­betes, the high glu­cose lev­els result from the fail­ure of the pan­creas to pro­duce the hor­mone insulin. In cas­es of severe insulin short­age, the body goes into a state called dia­bet­ic ketoaci­do­sis, which is a med­ical emer­gency.

Ear­ly on, doc­tors noticed that peo­ple with type 2 dia­betes rarely get dia­bet­ic ketoaci­do­sis. In severe cas­es, they might go into a coma because their blood sug­ar is so high that they get severe­ly dehy­drat­ed, but they rarely get such severe keto­sis that their blood pH is affect­ed. That’s because peo­ple with type 2 dia­betes are pro­duc­ing some insulin. In fact, they tend to have abnor­mal­ly high lev­els of insulin in their blood­stream. The prob­lem is that their cells have become resis­tant to the effects of insulin.

MODY was dis­cov­ered in the 1960s, when physi­cians noticed that some chil­dren had per­sis­tent, symp­tom­less high blood sug­ar that didn’t progress to ordi­nary type 1 dia­betes. In oth­er words, the con­di­tion looked like type 2 dia­betes, except that it was hap­pen­ing in young peo­ple who weren’t over­weight. The child’s pan­creas was pro­duc­ing some insulin, just not enough to meet the body’s needs. By the 1970s, it was clear that this prob­lem ran in fam­i­lies. At present, at least 10 forms of MODY have been described, each of them linked to a dif­fer­ent gene.

The treat­ment for MODY depends on what type of MODY the patient has. Genet­ic test­ing is now avail­able for MODY1, MODY2, MODY3, MODY4, and MODY5. MODY2 is a rel­a­tive­ly mild dis­ease that can be man­aged by diet and exer­cise. As in ordi­nary cas­es of type 2 dia­betes, the goal is to avoid or reverse insulin resis­tance, so that the amount of insulin the pan­creas pro­duces on its own will be enough to reg­u­late the patient’s blood sug­ar nat­u­ral­ly. Main­tain­ing insulin sen­si­tiv­i­ty through prop­er diet and exer­cise is undoubt­ed­ly also impor­tant in man­ag­ing the oth­er, more severe forms of MODY. Patients with MODY1, MODY3, or MODY4 are usu­al­ly giv­en a sul­fony­lurea drug that caus­es the pan­creas to secrete extra insulin. MODY5 is a severe con­di­tion that is usu­al­ly treat­ed as if the patient has no pan­creas what­so­ev­er; the patient is giv­en insulin injec­tions as well as diges­tive enzymes that are tak­en by mouth.

Even­tu­al­ly, doc­tors found that a few new­borns had a con­di­tion that seemed like type 1 dia­betes. This con­di­tion, called neona­tal dia­betes, is gen­er­al­ly inher­it­ed as an auto­so­mal reces­sive trait. To get a case of infan­tile dia­betes, the baby has to inher­it a defec­tive ver­sion of a gene from both par­ents.

If you think that you or some­one in your fam­i­ly has either MODY or neona­tal dia­betes, con­tact the Nation­al Cen­ter for Mono­genic and Neona­tal Dia­betes at the Uni­ver­si­ty of Chica­go.


Behind Barbed Wire_PrintNote: For more infor­ma­tion about dia­betes in gen­er­al and the genet­ic forms of dia­betes in par­tic­u­lar, see my book Thin Dia­betes, Fat Dia­betes: Pre­vent Type 1, Cure Type 2.

If You’re Eating for Two, Why Are You Vomiting?

One of the most com­mon symp­toms of ear­ly preg­nan­cy in human beings is nau­sea and vom­it­ing. Why do so many preg­nant women have so much trou­ble keep­ing food down at the very time that their need for calo­ries and oth­er nutri­ents has just gone up? Why is this prob­lem com­mon in women but seem­ing­ly nonex­is­tent in preg­nant females of oth­er species? Is there some­thing wrong with the design of human preg­nan­cy, or is there some­thing wrong with the food the preg­nant woman is eat­ing? I’m inclined to sus­pect the food, espe­cial­ly because morn­ing sick­ness is com­mon in the Unit­ed States but rare to nonex­is­tent in soci­eties whose sta­ple foods all come from plants.

Vom­it­ing is a pow­er­ful defense mech­a­nism. It effec­tive­ly removes tox­ins and infec­tious agents from the stom­ach and even the upper intestines. It’s nature’s way of expelling things that shouldn’t be allowed to enter the body. This defense mech­a­nism may be par­tic­u­lar­ly impor­tant dur­ing preg­nan­cy. Stud­ies have con­sis­tent­ly shown that women who vom­it dur­ing ear­ly preg­nan­cy are less like­ly to have a mis­car­riage than are those who mere­ly feel nau­se­at­ed. Per­haps it’s because the vom­it­ing pre­vent­ed things that would be harm­ful dur­ing ear­ly preg­nan­cy from enter­ing the woman’s body. Thus, it’s prob­a­bly no coin­ci­dence that the nau­sea and vom­it­ing asso­ci­at­ed with preg­nan­cy tend to be most severe dur­ing the first trimester, which is the most sen­si­tive phase of devel­op­ment.

In a clas­sic arti­cle, Samuel Flax­man and Paul Sher­man explained how morn­ing sick­ness could end up pro­tect­ing the moth­er and the preg­nan­cy. They argued that morn­ing sick­ness is most com­mon when the major organ sys­tems are devel­op­ing, and the vom­it­ing seems to be trig­gered by the foods and bev­er­ages that are most like­ly to be harm­ful to the moth­er and the preg­nan­cy. Flax­man and Sher­man point­ed out that in 9 out of 9 stud­ies, women who expe­ri­enced morn­ing sick­ness were much less like­ly to mis­car­ry.

Flax­man and Sher­man not­ed that many preg­nant women have aver­sions to alco­holic and non­al­co­holic (main­ly caf­feinat­ed) bev­er­ages and strong-tast­ing veg­eta­bles, but the great­est aver­sions were to meats, fish, poul­try, and eggs. The impor­tance of ani­mal-based foods in caus­ing morn­ing sick­ness also became obvi­ous in a cross-cul­tur­al com­par­i­son. Sev­en soci­eties that were free of morn­ing sick­ness were sig­nif­i­cant­ly less like­ly to have ani­mal foods as dietary sta­ples and were sig­nif­i­cant­ly more like­ly to have only plants (main­ly corn) as sta­ple foods than were 20 soci­eties in which women expe­ri­ence morn­ing sick­ness.

Food­borne infec­tious or par­a­sitic dis­ease could be a seri­ous threat to the health of a preg­nant woman or her preg­nan­cy. Dur­ing preg­nan­cy, a woman’s immune sys­tem is already some­what sup­pressed, to keep it from attack­ing the preg­nan­cy. As a result, preg­nant women are more like­ly to catch seri­ous, poten­tial­ly dead­ly infec­tions. Infec­tious and par­a­sitic dis­eases are also a major threat to the devel­op­ing embryo. For exam­ple, if a preg­nant woman catch­es Tox­o­plas­ma, which is a par­a­site found in cat drop­pings or under­cooked beef, the par­a­site infec­tion could cause mis­car­riage, still­birth, or severe birth defects.

A preg­nant woman can pro­tect her health and her preg­nan­cy by sim­ply avoid­ing the foods that are like­ly to make her vom­it. A pure­ly plant-based diet pro­vides all of the nutri­ents that a preg­nant woman needs, except for vit­a­min D (which she can get from sun­shine) and vit­a­min B12 (which is made by bac­te­ria and is avail­able in a nice, clean tablet).

Pho­to by Tip­sTime­sAd­min

Why Do Chimpanzees Eat Meat?

Chim­panzees eat meat for two sim­ple rea­sons: they can catch it and they like it. Chim­panzees are par­tic­u­lar­ly like­ly to eat meat dur­ing the dry sea­son, when short­ages of the foods that nor­mal­ly make up the bulk of theirdi­et cause them to lose weight. Although the meat may be a use­ful source of calo­ries dur­ing the dry sea­son, wild chim­panzees don’t need to include meat or any oth­er ani­mal-based food in their diet to ful­fill their needs for pro­tein or any of the amino acids. In fact, plants pro­vide all of the nutri­ents that are known to be essen­tial for a chim­panzee, except for vit­a­min D (which they get from the abun­dant sun­shine in Africa) and vit­a­min B12 (which comes from bac­te­ria).

Many peo­ple think that I am sil­ly for ask­ing where goril­las get their pro­tein. They tell me that I should talk about chim­panzees instead. Often, they inform me that chim­panzees are far more sim­i­lar to human beings than goril­las are, as if I couldn’t tell that just by look­ing. These peo­ple are miss­ing my point: goril­las are the largest and most pow­er­ful liv­ing pri­mate and yet are the clos­est to fol­low­ing what human beings would con­sid­er a veg­an diet. Chim­panzees and human beings don’t need to eat meat to grow up big and strong because goril­las grow up to be far big­ger and stronger with­out it. Lawyers may rec­og­nize this as an a for­tiori argu­ment.

If a male goril­la, whose diges­tive sys­tem is prac­ti­cal­ly iden­ti­cal to a human being’s, can get enough pro­tein from veg­eta­bles to grow to weigh more than 400 pounds and be ten times as strong as a man, why shouldn’t I expect that a rel­a­tive­ly puny human Olympic weightlifter could also get enough pro­tein from a plant-based diet? My intent is to ridicule the Four Food Groups dog­ma that I was taught in sixth grade.

Goril­las don’t hunt or fish, and they don’t keep cows or chick­ens. As a result, they don’t eat meat or fish, dairy prod­ucts or eggs. The only ani­mal-source food they eat is “the oth­er, oth­er white meat”: ter­mites, slugs, and oth­er creepy-crawlies. These foods would make an insignif­i­cant con­tri­bu­tion to the goril­las’ pro­tein intake, which is already high because pro­tein accounts for a high per­cent­age of the calo­ries in leaves.

Bugs and slugs could be a use­ful source of vit­a­min B12, a micronu­tri­ent that is made by bac­te­ria in their intestines. Vit­a­min B12 is also pro­duced by bac­te­ria in a primate’s gas­troin­testi­nal tract. How­ev­er, the vit­a­min may be pro­duced so far along in the intesti­nal tract that it isn’t absorbed effi­cient­ly. No plants make vit­a­min B12, but goril­las and chim­panzees can prob­a­bly get enough vit­a­min B12 from the bac­te­ria in the bugs they eat and in the dirt that clings to their food. Plus, apes are not metic­u­lous about wash­ing their hands, if you get my drift. If you are wor­ried about get­ting enough vit­a­min B12, you don’t have to eat dirt or bugs. You can get it in a nice, clean tablet instead.

I don’t ask where chim­panzees get their pro­tein because chim­panzees do eat some meat. Chim­panzees prob­a­bly eat less meat than just about any human pop­u­la­tion oth­er than Bud­dhist monks. Nev­er­the­less, many peo­ple want to use chim­panzees’ meat con­sump­tion as an excuse for humans to eat meat.

The fact that chim­panzees’ meat con­sump­tion is large­ly sea­son­al goes far toward explain­ing why human beings have always eat­en meat. Chim­panzees are most like­ly to eat meat dur­ing the time of year when they are los­ing weight because their usu­al foods are in rel­a­tive­ly short sup­ply. Peo­ple think of meat as a source of pro­tein, but it’s main­ly a source of calo­ries, espe­cial­ly from fat. Meat is also a good source of sodi­um, which is in rel­a­tive­ly short sup­ply in the chim­panzees’ fruit and veg­etable diet.

The fact that chim­panzees eat the most meat dur­ing times of food short­ages sug­gests that their food choic­es fol­low a pat­tern that biol­o­gists call opti­mal for­ag­ing the­o­ry. Ani­mals try to get the most calo­ries for the least effort and with­out get­ting hurt. Opti­mal for­ag­ing the­o­ry explains why chim­panzees eat meat but goril­las don’t, and why chim­panzees eat more meat dur­ing times of food short­age.

Chim­panzees are main­ly fruit eaters, but they also eat a lot of veg­eta­bles. The prob­lem with fruit is that it’s sea­son­al. Worse yet, a fruit tree rep­re­sents a rich enough source of calo­ries that ani­mals will fight over it. When fruit is scarce, chim­panzees can use the skills they devel­oped in fight­ing over the fruit to engage in preda­to­ry behav­ior. Also, chim­panzees are small enough and fast enough that they are rea­son­ably good hunters.

Goril­las, on the oth­er hand, main­ly eat leaves. There are gen­er­al­ly plen­ty of leaves to go around, and a leafy plant is gen­er­al­ly so poor in calo­ries that it’s not worth fight­ing to pro­tect. To sub­sist on leaves, how­ev­er, you have to eat an enor­mous vol­ume of food. Since leaves are so low in calo­ries, leaf-eaters have to be good at con­serv­ing their ener­gy. That’s why goril­las have such a placid dis­po­si­tion. For a goril­la, hunt­ing is sim­ply not worth the effort. They are too big and slow to catch very much, and they’re large enough that they’d risk injury if they got too reck­less.

Chim­panzees use twigs to fish for ter­mites, and goril­las don’t. Some peo­ple think that this fact means that chim­panzees are smarter than goril­las. I don’t. If you are a juve­nile goril­la or a preg­nant or nurs­ing female goril­la, you don’t need to mess around with a lit­tle bit­ty twig to get a few ter­mites. All you have to do is wait for the sil­ver­back to knock over a rot­ting tree. Then all of you can eat as many ter­mites as you’d like.

Some peo­ple have argued that the bal­ance between ani­mal and plant foods in a hunter-gath­er­er society’s diet rep­re­sents the opti­mal bal­ance for human nutri­tion. I think that’s idi­ot­ic. Hunter-gath­er­er peo­ples (or should I say, gath­er­er-hunter peo­ples) tend to fol­low opti­mal for­ag­ing the­o­ry just like any oth­er oppor­tunis­tic feed­er. Their goal is to sur­vive in the short term, not to avoid breast or prostate can­cer in mid­dle or old age. The main threat to their short-term sur­vival is star­va­tion.

Meat rep­re­sents a con­cen­trat­ed source of calo­ries. The fact that a rel­a­tive­ly high per­cent­age of these calo­ries comes from pro­tein is actu­al­ly a dis­ad­van­tage. Hunt­ing peo­ples pre­fer the fat­ti­est foods. Peo­ple who end up hav­ing to sub­sist on extreme­ly low-fat meat, such as rab­bit, are prone to a prob­lem called fat-hunger or rab­bit star­va­tion. This prob­lem prob­a­bly results from a diet that has too much pro­tein and not enough car­bo­hy­drate or fat. On a low-carb diet and dur­ing star­va­tion, the body has to make its sug­ar sup­ply out of pro­tein. Per­haps the body can make only so much sug­ar out of pro­tein. As long as you are eat­ing enough fat to meet most of your ener­gy needs, your body can make enough sug­ar out of pro­tein to feed your brain. If you were eat­ing pro­tein but not enough fat or carbs, you could end up in seri­ous trou­ble. So you could end up in trou­ble from a diet that is too high in pro­tein. In con­trast, it is prac­ti­cal­ly impos­si­ble to avoid get­ting enough pro­tein, as long as you are eat­ing enough unre­fined plant foods to get enough calo­ries.

Famine is not a sig­nif­i­cant cause of death in the Unit­ed States. In fact, peo­ple in the Unit­ed States are far more like­ly to die of the dis­eases of afflu­ence, such as heart dis­ease and can­cers of the breast and prostate. Ani­mal-based foods and fat­ty processed foods are the main con­tribut­ing caus­es of the dis­eases of afflu­ence. The abil­i­ty to use ani­mals for food may have helped human beings sur­vive to the mod­ern era, espe­cial­ly in the Arc­tic, but ani­mal-based foods are a major cause of death and dis­abil­i­ty in the Unit­ed States today. Think about that the next time you hear some­one pro­mot­ing a “Paleo” diet.

A Fish Is Not a Vegetable!

Late­ly, lots of peo­ple have been claim­ing that seafood is an impor­tant part of a health-pro­mot­ing diet for human beings. Some of the hype comes from the seafood indus­try, and some of it comes from peo­ple who sim­ply want an excuse to eat seafood. In real­i­ty, the health ben­e­fits of the so-called pesc­etar­i­an diets (a veg­e­tar­i­an diet plus seafood) result from the fact that they include a lot more starch and veg­eta­bles than is cus­tom­ary in the stan­dard Amer­i­can diet, while exclud­ing some of the most dan­ger­ous ani­mal-based foods. The starch and veg­eta­bles are good for you. Avoid­ing meat and milk from mam­mals and meat and eggs from birds is good for you. Unfor­tu­nate­ly, the wine and seafood and olive oil in the “Mediter­ranean” diet do more harm than good.

It has always struck me as illog­i­cal for peo­ple to call them­selves veg­e­tar­i­an if they eat seafood, which is the gen­er­al term used to include edi­ble fish and shell­fish. (Yes, there are some edi­ble plants that grow in sea­wa­ter, but they’re gen­er­al­ly called sea veg­eta­bles rather than seafood.) Fish are not veg­eta­bles. They are ani­mals. So are shell­fish, a cat­e­go­ry that includes mol­lusks such as oys­ters and crus­taceans such as shrimp and lob­ster. If you are eat­ing ani­mals, you’re not veg­e­tar­i­an.

Many peo­ple eat fish because they are afraid that a pure­ly plant-based diet wouldn’t pro­vide enough pro­tein to main­tain their health. That’s non­sense. Pro­tein defi­cien­cy is sim­ply not a real con­cern. As long as you get enough calo­ries from any prac­ti­cal diet based on unre­fined plant foods, you will auto­mat­i­cal­ly get enough protein—unless you have some bizarre diges­tive or meta­bol­ic dis­ease.

Rather than wor­ry­ing about not get­ting enough pro­tein, most peo­ple should be wor­ried about the effects of eat­ing too much pro­tein. When you eat more pro­tein than you need, your body turns the excess amino acids to sug­ar, releas­ing tox­ic waste prod­ucts such as ammo­nia and sul­fu­ric acid. In con­trast, burn­ing car­bo­hy­drates and fats for ener­gy pro­duces just car­bon diox­ide and water. The tox­ic byprod­ucts of a high-pro­tein diet can harm the liv­er and kid­neys, as well as pro­mot­ing osteo­poro­sis. One study showed that peo­ple from the North Slope of Alas­ka had high rates of bone loss as a result of their high-pro­tein diet, even though their cal­ci­um intake was high because they were eat­ing fish bones.

Seafood is ani­mal tis­sue, and it has the same faults as any oth­er ani­mal tis­sue. It con­tains cho­les­terol, too much pro­tein and fat, and no starch or fiber. Fish and oth­er sea crea­tures don’t pro­vide any essen­tial nutri­ents that you can’t eas­i­ly get from oth­er sources. Plants con­tain all of the nutri­ents that are essen­tial in human nutri­tion except for vit­a­min D (which you get from sun­shine) and vit­a­min B12 (which comes from bac­te­ria). Even the omega 3 fat­ty acids in fish oil came from the plants that were at the bot­tom of the fish’s food chain.

Anoth­er prob­lem with ani­mal tis­sue, includ­ing seafood, is the buildup of tox­ic sub­stances, includ­ing heavy met­als and fat-sol­u­ble chem­i­cals such as diox­in. This prob­lem is called bioac­cu­mu­la­tion. The high­er up in the food chain an ani­mal is, the worse this prob­lem tends to be. You can avoid this prob­lem by eat­ing plants instead of ani­mals.

In short, the hype about a “pesc­etar­i­an” diet is just hype. Peo­ple are bet­ter off just eat­ing plants.

Pho­to by Pardee Ave.